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匿名使用者
匿名使用者 發問時間: 社會與文化語言 · 1 0 年前

請問這篇關於吸菸的paper摘要在說什麼?

因為我爸常抽煙,我找了一篇來看,但是看不太懂,是否可以解釋給我聽呢?

感激不盡!!!!

題目:(Effects of cigarette smoking on blood antioxidant status in short- term and long-term smokers)

Abstract: To determine the effects of cigarette smoking on blood antioxidant status and lipid peroxidation, 16 healthy male current smokers (CS) and 16 healthy male non-smokers (NS) serving as controls were studied. CS were divided into two groups: short-term smokers (STS) 35.4 ± 5.8 years of age (mean±SD), and long-term smokers (LTS) 60.9 ± 4.9 years of age. The average moking period of STS and LTS were 8.1 ±1.1 years (mean ±SD) and 20.5 ± 4.5 years (mean ± SD)respectively. STS and LTS had their own controls, who were NS (young NS and old NS respectively).

When STS and LTS values are compared with their control values, the following are determined: 1-SOD and catalase activities increased significantly (p<0.05) in LTS, but were unchanged in STS. 2-STS and its control contained the same quantity of total glutathione and reduced glutathione (GSH). However, the oxidized glutathione (GSH). However, the oxidized significantly elevated (p<0.01). 3- In LTS, reduced glutathione diminished (p<0.01), while GSSG increased significantly (p<0.01), but total GSH was unchanged. 4-Smoking did not affect a-tocopherol level in STS, but an apparent decrease was observed in LTS (p<0.05). 5-Thiobarbituric acid reactive substance (TBARs) level as an index of lipid peroxidation increased significantly (p<0.05) in LTS, in increased significantly (p<0.05) in LTS, in 6-In comparing short-term and long-term smokers to each other, significant decreases were observed in total glutathione, reduced glutathione and a -tocopherol levels (p<0.05) in LTS. There were no significant differences between STS and LTS in the other measured parameters. It was concluded that the changes in the enzymatic and nonenzymatic antioxidant defense systems of elderly smokers may be due to oxidative stress caused by cigarette smoking. Key Words: Oxidative stress, antioxidants, TBARs

2 個解答

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  • 匿名使用者
    1 0 年前
    最佳解答

    這篇其實不難,上面那一個只是用翻譯軟體翻一下,很多意思都不對!例如 裡面的catalase是一種在酵素,在人體內會將H2O2反應變成水跟氧,但他卻翻成草地= ="

    建議你這種東西要自己翻,真的不懂來信問我, 我再跟你說吧!!!

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  • 匿名使用者
    1 0 年前

    在短期和長期的吸煙者裡的吸煙對血抗氧化劑地位的影響)

    摘要︰ 確定吸煙對血抗氧化劑地位的影響和類脂peroxidation, 16位健康的男性當今的吸煙者(CS)和16作為控制的健康的男性非吸煙者的(毫微秒)被研究。 CS 被分成兩組︰ 短期吸煙者(聖)35.4∮5.8歲(mean∮SD),和長期吸煙者(LTS)60.9∮4.9歲。 聖和LTS的平均moking 時期在4.5 年分別(想∮為南達科他)在1.1 年(意味著∮SD)和20.5∮是8.1∮。 聖和LTS有他們自己的控制,是毫微秒(分別的年輕的毫微秒和老的毫微秒)。

    當聖和LTS價值與他們的控制價值相比較時,下列被確定︰ 1草地和catalase 活動顯著增加(在LTS裡便士<0.05),但是在聖裡不變。 2聖和它的控制包含相同的總glutathione的數量並且降低glutathione(GSH)。 不過,被氧化的glutathione(GSH)。 不過,被使氧化的相當提升的(便士<0.01)。 3-在LTS內,降低glutathione 減少(便士<0.01),而GSSG 相當增加(便士<0.01),但是總GSH不變。 吸煙4在街沒影響一tocopherol 水準,但是明顯的減少被用LTS 觀察(便士<0.05)。 一個類脂peroxidation的索引顯著增加的5-Thiobarbituric酸無功的物質(TBARs)水準(在LTS裡的便士<0.05), 在方面增加相當(便士<0.05)在內LTS, 在在6過程中的把短期和長期的吸煙者比作彼此,顯著的減少是被全部觀察的glutathione,降低glutathione 和A -tocopherol 水準(在LTS裡的便士<0.05)。 在聖和在其它合標準的參數裡的LTS之間沒有明顯的不同。 斷定在老吸煙者的 催和nonenzymatic 抗氧化劑防禦系統方面的變化可能由於透過吸煙被引

    參考資料: DR.EYE
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